Note:  This particular Source Reading was hard to edit and will be hard to read.  It goes into deep medical detail of what the Source views as the cause of AIDS.

. . . Our view or description of this disease process does not agree, in some specifics, with current conventional understanding.   We have a concern here for a bacterium that, in some sense, is more dangerous than the specified virus.  We would like to comment in this way: It will eventually be discovered that it is the nature of a virus to be produced within a body as a result of a prior imbalance.

Now we make that point to establish this: bacteria are commonly carried from body to body through various means of communication.  Now living virus can also be carried from body to body, but are created within the body of the receiver only in the presence of specific conditions which allow for the creation of the virus.

That is to say, viral infections are not carried or communicated in the same manner as bacteria, but are created within that body under conditions of an immune system which is already under attack. Virus transferred from body to body is, in most cases, not viable. That is, virus is a protein chain specific to the body in which it is created and encountering another body, is simply dissolved. To say that it is killed is an inappropriate term for the reason that it suggests a living organism, as is for example bacteria, which virus is not, but rather is a specific protein chain formed in reaction to pre-existing conditions.

Now in the presence of the communication of a bacterial strain which we would describe as mutated, there has been produced a mutated bacterium that is common to what was a strain of syphilis, including the existence of syphilis specific spirochetes.

Now, the bacteria is not recognizable as such, at this point, and it is not to suggest that one infected with the immune suppressant virus has already had or previously carried syphilis. It does mean that the carrier does carry a mutant bacterium, which in fact, becomes a mutant gene.  You will find this, unfortunately, as it carries forward in the further development of this disease.   So that it becomes a genetically coded immune suppressed system, unless there is recognition of the symbiotic relationship between bacteria and virus involved in the attack on the immune system.

We say all of these things for this purpose: there are attempts to repress or destroy a virus which has been identified and associated with suppressed immune systems. However, there is not a distinct relationship between the suppression of the immune system and the existence of the virus. What is rather discovered is that they co-exist.

The inference of the co-existence has lead researchers to assume that the co-existent virus is the cause specific of the immune suppression. With this one point we disagree to this extent: without the combination of three elements co-existing, there is not the acquired immune deficiency syndrome. Not a disease specific, but simply an acquired immune suppression. There is not an established disease caused by the HIV virus or other names it has been given. You do rather have these elements present in the production of the immune suppression.

At one point in one body, there is present the infection referred to as syphilis.   Within that body, there are introduced antibiotics, antibiotics insufficient to destroy all  destructive bodies, related to and specific to syphilis. These we refer to as bacteria and spirochetes. This condition being treated (with antibiotics) to the extent of the suppression of symptoms, whether temporarily or permanently, have, for some time, been administered in amounts insufficient to totally eradicate the presence of the spirochetes, in particular, and the associated bacteria has mutated to become largely immune to antibiotics.

In that same system currently spreading as an epidemic referred to as acquired immune deficiency syndrome, those bodies in which these combinations of forces were created, had present a syphilis infection.  (Thus) creating spirochetes and bacteria in the presence of antibiotics, which created a yeast bacterium imbalance, making the yeast relationship to bacteria imbalanced.  Thus allowing yeast infections, which are difficult to detect and are opportunistic (and are) associated with the mutant bacterium, and eventually, the gene developing from the presence of the yeast/bacterium/spirochete/ virus relationship.

It is a strange combination of forces which has not before been encountered on your planet.  For without the presence of one of these bodies, the others would eradicate one another.  That is to say: the virus allows the yeast to live, even without eradicating or re-creating balance between the exceptionally resistant bacterium.   This results in an unusual phenomena of elevated yeast count, along with and simultaneous to, an elevated bacteria count. So that the result is not recognized as an opportunistic yeast infection, though that, in fact, is a part of the syndrome. And the presence of the unrecognized bacterium is only now being discovered to be associated with the forces.

All of this we have (presented as cause is to offer this suggested treatment): you do have sufficiently powerful antibiotics, which used in extraordinary dosages, so high that your practitioners are reluctant to administer them, for good reason. That reason being the creation of a difficult, opportunistic yeast imbalance in the body and other imbalances often not associated with the growth of yeast.

A difficulty here is simply that the growth of yeast within the body is considered natural and is not yet fully recognized as a syndrome on its own, which is a direct result of the discovery and use of antibiotics.

Then the use of antibiotics in the presence of imbalanced yeast would seem to further advance the opportunistic nature of the yeast infection, and so it would. Thus we have a complex situation in which exceptionally strong high doses of antibiotics should be administered in situations where the body is being obviously defeated or overwhelmed by the advancement of the immune deficient syndrome.

However, it must be balanced by an anti-yeast antibiotic. (This must be done) either simultaneously with the administration of the antibiotic, or subsequent to the sufficiently high introduction of antibiotics, bacterium-specific, and perhaps sufficiently broad-spectrum to administer yeast control as well. Though they do not have to be introduced together, the antibiotic can be bacteria-specific first, then yeast-specific following.

Now, such applications of penicillin or other forms of bacteria-specific antibiotics will in fact conquer both the symptoms related to immune deficiency syndrome.  And, with the yeast imbalance also brought under control, the delicate balance between these two natural organisms within the body (will be maintained).

Let us be clearer with that statement.  We are saying that both yeast and bacteria exist naturally within the human body.  However, yeast out of balance or (out of) control is not natural to the body.  It is further not natural that a bacterium can take effective control of a yeast-overwhelmed body, for these are natural enemies in that they tend to balance one another. This imbalance is tampered with in the introduction of bacteria-specific antibiotics, allowing the growth of various yeast and molds, but yeast in particular. Thus there is the difficult challenge of bringing both into control and balance.

The confusing factor is that the mutant bacteria is not killed by the over-production of yeast as bacteria ordinarily are. This is because the bacteria has mutated as a result of having been treated over several generations with a yeast-based antibacterial, you see. Yeast based antibiotics were used to kill a bacterium, but were used in insufficient amounts, which allowed the bacterium (to mutate) and, in most cases of that you refer to as acquired immune deficiency, that specific bacterium is related to syphilis.  It has thus become immune to a large extent, to yeast-based, or yeast derived antibiotics. Thus it can grow, even in the presence of a high yeast count.

Now these are things that must be discovered, investigated, researched and observed. And as these facts are observed, you will find that we have correctly described the development of this syndrome.

Continued in Part 3